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Together, these findings demonstrate: (i) modulatory effects of CD38 mAbs on ADPR-Cyclase and cADPR-Hydrolase activities in the LP-1 line, and (ii) antibody-specific differences in regulating CD38 catalytic functions

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These findings suggest that Dihexa could be a potential treatment for Alzheimer's disease [3]

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Mitochondrial potential loss was measured using the Varioskan kinetic plate reader at excitation 545 nm and emission 590 nm for 3 h at 21 C (kinetic measurements every 5 min)

Schubert-Bast and Strzelczyk, 2021)