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Clarkson, B, Gee T, Arlin Z, Mertelsmann R, Kempin S, Dinsmore R, O'Reilly R, Andreeff M , Berman E, Higgins C, Little C, Cirrincione C, Ellis S

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[DOI] [PubMed] [Google Scholar] 150.Labb A., Brignole-Baudouin F., Baudouin C

Reactive oxygen species, cytokines, and axonal damage in multiple sclerosis Mechanisms of axonal damage are the consequence of the presence of TNF, matrix metalloproteinases (MMPs), ROS, antibodies, increased glutamate, and aspartate, and these molecules cause excitotoxicity in MS patients

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Further studies have shown the absence of GSH in T cells does not interfere with TCR signaling but leads to a failure to reprogram cellular processes needed for robust clonal expansion and prevents the induction of aerobic glycolysis and the increase in cell size associated with T cell activation (Klein Geltink et al., 2017)