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By pairing up with a complementary host mRNA sequence, the viral antisense strand could prevent translation of the host mRNA, thus suppressing host selenoprotein gene expression (Fig

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[6] In the second step, 2,3-bisphosphoglycerate is dephosphorylated into 3-phosphoglycerate

Since this pilot study, a larger group of DOA patients carrying either OPA1 mutations leading to haploinsufficiency or missense point mutations has been treated with idebenone [14]